Placental mitochondrial function as a driver of angiogenesis and placental dysfunction

Keyphrases

• Mitochondrial Function 100%
• Angiogenesis 100%
• Placental Dysfunction 100%
• Soluble Fms-like Tyrosine kinase-1 (sFlt-1) 75%
• Mitochondria 50%
• Antiangiogenic Factors 50%
• Dysregulation 50%
• Mitochondrial Adaptation 50%
• Reactive Oxygen Species 25%
• Blood Vessels 25%
• Oxidative Stress 25%
• Environmental Change 25%
• Stillbirth 25%
• Vascularized 25%
• Angiogenic Factors 25%
• Preeclampsia 25%
• Preeclamptic 25%
• Placenta 25%
• Selective Intrauterine Growth Restriction (sIUGR) 25%
• Placental Angiogenesis 25%
• Placentation 25%
• Nutrient Transport 25%
• SFlt 25%
• Healthy Pregnancy 25%
• Fetal Development 25%
• Required Energy 25%
• Hormone Synthesis 25%
• Oxygen Use 25%
• Nutrient Use 25%
• Fetal Organs 25%

Medicine and Dentistry

• Angiogenesis 100%
• Placenta Disorder 100%
• Angiogenic Factor 75%
• Mitochondrion 50%
• Antiangiogenic 50%
• Oxidative Stress 25%
• Reactive Oxygen Species 25%
• Stillbirth 25%
• Pre-Eclampsia 25%
• Placenta 25%
• Intrauterine Growth Retardation 25%
• Placenta Development 25%
• Fetus Development 25%
• Hormone Synthesis 25%
• Fetal Organ 25%

Biochemistry, Genetics and Molecular Biology

• Angiogenesis 100%
• Mitochondrion 50%
• Oxidative Stress 25%
• Prenatal Growth 25%
• Placenta Development 25%
• Fetus Development 25%
• Hormone Synthesis 25%