TNF-α-Mediated Endothelial Cell Apoptosis Is Rescued by Hydrogen Sulfide

Keyphrases

• Caspase-3 100%
• Endothelial Dysfunction 100%
• Hydrogen Sulfide 100%
• GYY4137 100%
• Endothelial Cell Apoptosis 100%
• Mitochondrial Health 100%
• Beneficial Effects 50%
• Endothelial Cells 50%
• Apoptosis 50%
• Caspase Activation 50%
• Cytochrome c Release 50%
• Diabetes 50%
• Cardiovascular Disease 50%
• Induced Apoptosis 50%
• Redox 50%
• Vasculature 50%
• Endothelium 50%
• Molecular Mechanism 50%
• Protective Effect 50%
• Redox State 50%
• Caspase-3 Activity 50%
• Regulatory Mechanism 50%
• Preeclampsia 50%
• Chronic Disease 50%
• Slow-releasing 50%
• H2S Donors 50%
• Vasoprotective 50%
• Gasotransmitter 50%
• Vascular Oxidative Stress 50%
• Aggravation 50%
• Cardiovascular Complications 50%
• Mitochondrial Morphology 50%
• S-sulfhydration 50%
• Mitochondrial Network 50%
• Mitochondrial Cytochrome c 50%

Pharmacology, Toxicology and Pharmaceutical Science

• Tumor Necrosis Factor 100%
• Hydrogen Sulfide 100%
• Endothelial Dysfunction 40%
• Procaspase 3 40%
• Cardiovascular Disease 40%
• Cytochrome C 20%
• Caspase 20%
• Anti-Inflammatory Drug 20%
• Bioavailability 20%
• Chronic Disease 20%
• Atherosclerosis 20%
• Preeclampsia 20%
• Cytoprotective Agent 20%
• Gasotransmitter 20%